mutations and trisomy twelve are connected with precise reworking of chromatin activation and accessibility areas. Far more specifically, the epigenomic profile induced by MYD88

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り当て制御を行えば，性能向上が見込めると考えられる． 理論計算とシミュレーションによる評価結果から，提案

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translocations or amplifications on top of the genomic alterations now current in the first CLL, but absence the common mutations noticed in Main DLBCL indicating which they might correspond to another biological group.

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